We have changed our domain
We have changed servers and operate Career|edu under a different domain. Our services will remain unchanged and we will continue to post the best career openings for the academic world at our new domain.
Go back to the Job List | ||
|
||
Post-translational Protein Modifications, Mitochondrial Dysfunction, Apoptosis and Tissue DamagePosted by: National Institutes of Health (NIH)Posted date: 2019-Jan-22
Location: USA
Position Description:
Postdoctoral fellow positions are immediately available to study the role of increased oxidative/nitrative stress in promoting post-translational protein modifications, mitochondrial dysfunction, ER stress, apoptosis and tissue damage caused by alcohol, drugs, fructose, and western-style high fat diets through the gut-liver-brain axis. Molecular biological, biochemical and proteomics methods are being used to study the mechanisms of alcohol- and high fat-induced organ damage (liver and brain damage) in various experimental models (cultured cells & transgenic or knockout mice) and human specimens. Qualifications:Applicants should have a Ph.D. and/or M.D. with less than five years of postdoctoral experience. A candidate with strong background in molecular and cell biology or protein biochemistry with in vivo animal experience is highly preferred. To Apply:Application: Highly motivated applicants should send their curriculum vitae with bibliography, a brief statement of research interest and career goals, and three reference letters (or contact information) to Dr. B. J. Song at bj.song NIH is an Equal Opportunity Employer. The NIH is dedicated to building a diverse community in its training and employment programs. |